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Ashraf A, Fan Z, Brooks DJ, Edison P. Cortical hypermetabolism in MCI subjects: a compensatory mechanism? Eur J Nucl Med Mol Imaging. September 2014.

Publication Date
September 2014

How Analyze was Used
” [¹¹C]PIB RATIO images were sampled using Analyze 11.0 in the following regions: frontal,
temporal, parietal and occipital association cortices, precuneus, anterior and posterior cingulate gyrus, hippocampus, parahippocampal gyrus, amygdala, striatum thalamus, and precentral and postcentral gyrus…. For the region of interest (ROI) analysis, rCMRGlc images of all individuals were spatially transformed into MNI space using SPM8 and sampled using Analyze 11.0 in the same regions as described for [¹¹C]PIB.”

Keywords
Amyloid-Negative
Amyloid-Positive
Compensatory Hypermetabolism
Mild Cognitive Impairment (MCI)
Neuroplasticity

Author Affiliation(s)
Neurology Imaging Unit, Division of Brain Sciences, Imperial College London, Block B Hammersmith Campus, Du Cane Road, London, W12 0NN, UK.

ID# 675

Tags: Amyloid-Negative, Amyloid-Positive, Compensatory Hypermetabolism, Mild Cognitive Impairment (MCI), Neuroplasticity